I was e-mailed by Roberta Seaman yesterday, to share with me her experience with a new product for lowering blood pressure and cholesterol.
Friday, September 17, 2010
Lower both your cholesterol and blood pressure naturally
I was e-mailed by Roberta Seaman yesterday, to share with me her experience with a new product for lowering blood pressure and cholesterol.
Just wanted to share it with you all.
Amr.
and it's heart health in a bottle, consisting of 19 fruitsI am not a fan of prescription drugs and wanted to try something all-natural, so I
Friday, May 21, 2010
Monday, May 17, 2010
Omega 3 fat myths busted and why you really need to have them as supplements
Spring Valley - Fish Oil Omega-3, 1000 mg, 200 Softgels (Twin-Pack 2 Bottles
Last month my over 50 year old diabetic uncle began to experience tingling in his feet. He’s the elder among my uncles and the one with the most responsibilities and visions concerning our family, so I guess his blood sugar level was rising too much for his regular medications for diabetes. Being a doctor, I went over to his office and confronted him that sometimes life issues can make us forget about ourselves, and our problems until, an issue like a disease, or a complication of a disease, which I consider a slap on the cheek from God, awakes us from our chosen sleep and makes us wonder about the importance of life.
We’ve found out that his fasting blood suger level was 275 mg/dl and that his glycosylated Hemoglobin was 13. Disasterous numbers to say the least. So, I’ve changed his diabetes medications and prescribed new medications for his numbing feet. Next week his fasting blood sugar level was surprisingly 110 mg. Days passed by, and his follow up tests became more reassuring, but his weight didn’t seem to increase in proportion to the ability of his body to benefit from his glucose, when I started thinking about omega 3 supplemets.
I knew his daily concerns, stresses , willingness to adhere to the prescription, weak teeth that won’t let him bite food as he did before, all will prevent him from gaining that much weight , so I began considering omega 3 supplements to provide him with the power of both body and mind and – at the same time – don’t elevate his blood sugar so much.
I began to read a lot about omega 3 supplements and what I’ve found was far more rewarding than I’ve ever expected. I can safetly consider giving it to my young children, my wife and my parents as well. I was also enlightened about another food ingredient that was hidden from me and many people all the past years although it’s wandering inside our bodies and may be causing us a lot of harm, and by that I mean THE OMEGA 6 FAT.
In fact the story behind the omega 6 fats dates far back to 1930 something, when it was discovered, and categorized by George and Mildred Burr at the university of Minnesota. Yet it was the first family of essential fats, and it was another 40 years before the omega 3s were also discovered and found to be essential by a researcher at Hormel named Ralph Holman. And while information was being discovered about those two families, some very interesting highlights began to be shed on how human health began to deteriorate over the years.
The first fact that was discovered was that the omega 3s and their in-laws, the omega 6s, were found primarily in plants, and that they don’t originate from fish as many people think ( a good news for people who are allergic to fish ofcourse ). Fish are full of omega 3s because they eat phytoplankton (the microscopic green plants found in lakes, rivers and oceans) together with seaweed. Within plants , these special omega 3 fatty acids help turn sunlight into sugars, the basis of life on earth.
Later, omega 3s were found to speed up metabolism, i.e. they are fats that animals and humans can use to get ready for times of activity. That was supported by the discovery of omega 3 fatty acids in high concentrations in all active tissues: brain, eyes, hearts, the tails of sperms, the flight muscles of the humming bird. Because fish had so many of these fats in their diets, they are active in cold dark waters.
But a question about the nature of omega fats in plants began to arise in concern to the cyclic life of most plants during the different seasons of the year, and the answer was surprising : Omega fats change with the seasons. Omega 3s are spring fats, while omega 6s are autumn fats. In light of the above two paragraphs, one can find an explanation of why omega 3s are spring fats and why they are located mainly in green leaves, but since omega 6s are fall fats, one can predict that they are concentrated more in plant seeds, the storage areas for renewal of life when conditions became better.
So, animals and humans take both omega families from plant with food, but the characteristics of each family of omega fats play a leading role in its effect on the human body. Omega 3 fats alone were found by studies to lower the incidence of blood clotting and inflammation, and also it accumulates in the brain and can help children with learning difficulties. They also accumulate and metabolcally active tissues, so it also lowers the risk for joint inflammation, cancer and help fight wrinkles and may block fat cell formation. A recent study in Harvard school of public health found that absence of these fatty acids from our diets are responsible for up to 96,000 premature deaths per year in England. On the other hand, omega 6 fatty acids promote blood clotting and inflammation, the milestone of many diseases.
The proper mix of these two fats help create tissue with the right amount of blood flow and inflammation, but because they are in constant competition to enter our cells, if your diet consists of too much omega 6s, your boby will be deficient in omega 3s. And that’s what has been happening to us as we’ve been eating more and more seed fats in the form of soybean, corn, and other vegetable oils. Since 1909, according to the USDA, Americans have more than doubled their daily intake of omega 6s from about 7 grams to around 18. Also, heart diseases has risen in tandem with our increasing intake of these seed fats, or omega 6s, according to the American Heart Association (AHA). Over the same time period, omega 3s began disappearing from our food supply. Cows used to be raised on grass and other greens, producing meat, milk and cheese with much higher concentrations of omega 3s. Now these livestock are fed on corn and soy, and their tissues are swamped with omega 6s. Chickens apply to that too. We are now eating a diet that is supposed to fatten us up for winter, so our supply has shifter from leaves to seeds, and this simple change means our bodies are storing more fat, leading to obesity and all its associated diseases.
So it’s important for us to correct that forced omega 3s deficiency by taking it as a supplement
Friday, May 14, 2010
Food is not your God anymore.....
Reductil ... What Really Works
Mechanisms for regulating food intake are still not clearly established. That the hypothalamus plays a part in these mechanisms, however, seems certain. Numerous studies seem to indicate that a cluster of neurons in the lateral hypothalamus function as an appetite center (meaning that impulses from them bring about increased appetite). Other data suggest that a group of neurons in the ventral medial nucleus of the hypothalamus functions as a satiety center (meaning that impulses from these neurons decrease appetite so that we feel sated, or satisfied. What acts directly on these centers to stimulate or depress them is still a matter of theory rather than a fact.
One theory ( the thermostat theory ) holds that it is the temperature of the blood circulating to the hypothalamus that influences the centers. A moderate decrease in blood temperature stimulates the appetite center (and inhibits the satiety center). Result : the individual has an appetite, wants to eat, and probably does. An increase in blood temperature produces the opposite effect, a depressed appetite (anorexia). One well-known instance of this is the loss of appetite in persons who have a fever.
Another theory ( the glucostat theory ) says that it is the glucose concentration and rate of glucose use that influences the hypothalamic feeding centers. A low blood glucose concentration and rate of glucose use stimulates the appetite center, whereas high blood glucose concentration inhibits it.
The use of (Reductil) as a drug for weight loss is highly linked to the body's own appetite control mechanisms, where it suppresses appetite an stimulates the body to use its own energy resources.
Buy at Best Generic Drugs
The active ingredient of (Reductil) is sibutramine. Sibutramine was originally developed as an antidepressant ( fights depression ), and it exerts its effect by helping two other substances: noradrenaline and serotonin stay as long as possible in the brain. Noradrenaline is the hormone of stress. It gushes through our blood at times of fight or fear moments to help us use all our bodily capabilities to either fight or run away from the stress at hand. I don't think anyone who might be reading that article ever had the desire to eat when faced by any threat. Serotonin is a local hormone that controls the release of another local hormone called leptin whose receptors are in the brain and their activation by leptin inhibits impulses that increase appetite, and stimulates impulses that decrease appetite.
This drug is rapidly absorbed from the gut, and it is extensively metabolized ( broken down ) in the liver, and its resulting substances have a half life ( time for the body to breakdown half of the substance ) is 14 - 16 hours, and is responsible for its effects. That's why (Reductil) should be taken early in the morning, so that it doesn't make you awake and disturb your sleeping time, especially on the first day of taking the drug.
Buy at Best Generic Drugs
(Reductil) shouldn't be taken carelessly by patients. It should be prescribed only for people with Body Mass Index (BMI) of 27 or more who have other heart or blood vessel risk factors, or 30 or more in their absence. It should be stopped if weight lost after 3 months is less than 5% of the initial weight. It should be stopped also if weight lost stabilizes at less than 5% of the initial weight thereafter ( despite increase in concentration of Reductil ), or if users regain more than 3 kg after previous weight loss. It should not be given for more than 1 year.
If one wanna calculate one's own BMI, Divide your weight in kilograms by the square of your height in meters. So for example, if your weight is 115 kg, and your height is 180 centimeters ( i.e. 1.8 meters ), so your BMI can be calculated as 115/(1.8)2 = 115/3.24 = 35.4, so, if you have no other contraindications to use (Reductil), you'll definitely be able to use it.
Being part of the antidepressant family, (Reductil) is not the drug which you might be drinking alcohol during its course of treatment, because both will exert a depressing effect on brain function. So try to be more conservative at birthday parties or celebrations please. The dose of (My Reductil) is 10-15 mg per day by mouth.
One of the most reported side effects experienced by users of (Reductil), and which - at the same time - are self-limited with continued use or - sometimes - symptomatic treatment, is lack of sleep especially in the first night or two of taking (Reductil), and the best way to handle that is by taking the pill very early in the morning.
Another very popular side effect, dry mouth, which occurs in more than 10% of users, but it's nothing to worry about. It may require the user to consume 2-3 litres of water a day, and always carry an extra bottle of water around, but to look at it from another optimistic view, drinking a lot of water daily actually fills the stomach and decreases appetite, so it's helpful anyway.
There is a famous forum on Yahoo about how good (Reductil) is as an anti-obesity drug. I've been to that forum reading people's feedback on that drug, and how it really helped most of them with their weight loss battles. Here are some of the remarkable quotes people said after sing that drug: " With My Reductil, I just have no interest in food ". One guy with inactive thyroid gland said: " Reductil is the only thing that helped me ". Another one quoted: " My Reductil stops you needing to eat. It gets rid of the hunger, but willpower needs to be strong ". A last one: " Food is not my God anymore ".
Buy at Best Generic Drugs
There is also one last relation between (Reductil), blood pressure and headache. That relation needs to be understood by patients in order not to be confused to the extent of quitting treatment for no reason. As I notified earlier, (Reductil) depends on noradrenaline and serotonin inside the brain to do what it does. As all would know, noradrenaline narrows blood vessels everywhere in the body which contributes to raising the blood pressure alittle, while serotonin, dilates blood vessels, and in the brain, may cause the headaches that (My Reductil) users may experience especially on the first days of treatment. The point is : Don't just quit (My Reductil) if your blood pressure rises a little bit. You can stop it if the rise in blood pressure is severe ( more than 180/110 ), so that's why doctors recommend that (Reductil) users should check their blood pressure closely throughout its use ( twice weekly during the first 3 months ). You shouldn't worry much about the headache because it goes away later, and as far as your blood pressure is fine, take a panadol or something or the headache and life will go on.
In fact, you should benefit from the 3 months that you start your (Reductil) in, and not rush losing weight too fast. Losing weight so fast may let you get saggy skin, or let your kidney fall off inside your abdomen due to quick loss of the fat pad attaching it to the wall of the abdomen, so those first 3 months should be used by you wisely. The first month is all about surviving the changes that taking (Reductil) will have on your life. The second month should be all about trying to establish an eating pattern. The third month should be all about introducing a more extensive exercise program.
The last thing to be mentioned here is that (Reductil) should not be used in ( severe ) increase in blood pressure. It should not be used at all with peripheral occlusive arterial or coronary heart disease, arrhythmias, enlarged prostate condition, and in severe liver or kidney disease. It shouldn't also be used to treat obesity of endocrine origin, or those with a major eating disorder or a psychiatric disease. It also shouldn't be taken together with a tricyclic antidepressant, because both may lead to central nervous system toxicity.
Buy at Best Generic Drugs
Mechanisms for regulating food intake are still not clearly established. That the hypothalamus plays a part in these mechanisms, however, seems certain. Numerous studies seem to indicate that a cluster of neurons in the lateral hypothalamus function as an appetite center (meaning that impulses from them bring about increased appetite). Other data suggest that a group of neurons in the ventral medial nucleus of the hypothalamus functions as a satiety center (meaning that impulses from these neurons decrease appetite so that we feel sated, or satisfied. What acts directly on these centers to stimulate or depress them is still a matter of theory rather than a fact.
One theory ( the thermostat theory ) holds that it is the temperature of the blood circulating to the hypothalamus that influences the centers. A moderate decrease in blood temperature stimulates the appetite center (and inhibits the satiety center). Result : the individual has an appetite, wants to eat, and probably does. An increase in blood temperature produces the opposite effect, a depressed appetite (anorexia). One well-known instance of this is the loss of appetite in persons who have a fever.
Another theory ( the glucostat theory ) says that it is the glucose concentration and rate of glucose use that influences the hypothalamic feeding centers. A low blood glucose concentration and rate of glucose use stimulates the appetite center, whereas high blood glucose concentration inhibits it.
The use of (Reductil) as a drug for weight loss is highly linked to the body's own appetite control mechanisms, where it suppresses appetite an stimulates the body to use its own energy resources.
Buy at Best Generic Drugs
The active ingredient of (Reductil) is sibutramine. Sibutramine was originally developed as an antidepressant ( fights depression ), and it exerts its effect by helping two other substances: noradrenaline and serotonin stay as long as possible in the brain. Noradrenaline is the hormone of stress. It gushes through our blood at times of fight or fear moments to help us use all our bodily capabilities to either fight or run away from the stress at hand. I don't think anyone who might be reading that article ever had the desire to eat when faced by any threat. Serotonin is a local hormone that controls the release of another local hormone called leptin whose receptors are in the brain and their activation by leptin inhibits impulses that increase appetite, and stimulates impulses that decrease appetite.
This drug is rapidly absorbed from the gut, and it is extensively metabolized ( broken down ) in the liver, and its resulting substances have a half life ( time for the body to breakdown half of the substance ) is 14 - 16 hours, and is responsible for its effects. That's why (Reductil) should be taken early in the morning, so that it doesn't make you awake and disturb your sleeping time, especially on the first day of taking the drug.
Buy at Best Generic Drugs
(Reductil) shouldn't be taken carelessly by patients. It should be prescribed only for people with Body Mass Index (BMI) of 27 or more who have other heart or blood vessel risk factors, or 30 or more in their absence. It should be stopped if weight lost after 3 months is less than 5% of the initial weight. It should be stopped also if weight lost stabilizes at less than 5% of the initial weight thereafter ( despite increase in concentration of Reductil ), or if users regain more than 3 kg after previous weight loss. It should not be given for more than 1 year.
If one wanna calculate one's own BMI, Divide your weight in kilograms by the square of your height in meters. So for example, if your weight is 115 kg, and your height is 180 centimeters ( i.e. 1.8 meters ), so your BMI can be calculated as 115/(1.8)2 = 115/3.24 = 35.4, so, if you have no other contraindications to use (Reductil), you'll definitely be able to use it.
Being part of the antidepressant family, (Reductil) is not the drug which you might be drinking alcohol during its course of treatment, because both will exert a depressing effect on brain function. So try to be more conservative at birthday parties or celebrations please. The dose of (My Reductil) is 10-15 mg per day by mouth.
One of the most reported side effects experienced by users of (Reductil), and which - at the same time - are self-limited with continued use or - sometimes - symptomatic treatment, is lack of sleep especially in the first night or two of taking (Reductil), and the best way to handle that is by taking the pill very early in the morning.
Another very popular side effect, dry mouth, which occurs in more than 10% of users, but it's nothing to worry about. It may require the user to consume 2-3 litres of water a day, and always carry an extra bottle of water around, but to look at it from another optimistic view, drinking a lot of water daily actually fills the stomach and decreases appetite, so it's helpful anyway.
There is a famous forum on Yahoo about how good (Reductil) is as an anti-obesity drug. I've been to that forum reading people's feedback on that drug, and how it really helped most of them with their weight loss battles. Here are some of the remarkable quotes people said after sing that drug: " With My Reductil, I just have no interest in food ". One guy with inactive thyroid gland said: " Reductil is the only thing that helped me ". Another one quoted: " My Reductil stops you needing to eat. It gets rid of the hunger, but willpower needs to be strong ". A last one: " Food is not my God anymore ".
Buy at Best Generic Drugs
There is also one last relation between (Reductil), blood pressure and headache. That relation needs to be understood by patients in order not to be confused to the extent of quitting treatment for no reason. As I notified earlier, (Reductil) depends on noradrenaline and serotonin inside the brain to do what it does. As all would know, noradrenaline narrows blood vessels everywhere in the body which contributes to raising the blood pressure alittle, while serotonin, dilates blood vessels, and in the brain, may cause the headaches that (My Reductil) users may experience especially on the first days of treatment. The point is : Don't just quit (My Reductil) if your blood pressure rises a little bit. You can stop it if the rise in blood pressure is severe ( more than 180/110 ), so that's why doctors recommend that (Reductil) users should check their blood pressure closely throughout its use ( twice weekly during the first 3 months ). You shouldn't worry much about the headache because it goes away later, and as far as your blood pressure is fine, take a panadol or something or the headache and life will go on.
In fact, you should benefit from the 3 months that you start your (Reductil) in, and not rush losing weight too fast. Losing weight so fast may let you get saggy skin, or let your kidney fall off inside your abdomen due to quick loss of the fat pad attaching it to the wall of the abdomen, so those first 3 months should be used by you wisely. The first month is all about surviving the changes that taking (Reductil) will have on your life. The second month should be all about trying to establish an eating pattern. The third month should be all about introducing a more extensive exercise program.
The last thing to be mentioned here is that (Reductil) should not be used in ( severe ) increase in blood pressure. It should not be used at all with peripheral occlusive arterial or coronary heart disease, arrhythmias, enlarged prostate condition, and in severe liver or kidney disease. It shouldn't also be used to treat obesity of endocrine origin, or those with a major eating disorder or a psychiatric disease. It also shouldn't be taken together with a tricyclic antidepressant, because both may lead to central nervous system toxicity.
Buy at Best Generic Drugs
Monday, May 10, 2010
Nine things to do if you think you have high cholesterol
When it comes to cholesterol, there is no normal cholesterol level. Instead, there is a cholesterol level that predict higher exposure to death and disease if deposited beneath the internal lining of our blood vessels. And there is a cholesterol value that is associated with less likelihood of heart and blood vessel disease.
Basic science studies have shown that deposition of cholesterol in arteries is an inflammatory disease. One of the key factors triggering this inflammation is LDL. When LDL is taken up by certain cells in the blood stream, it triggers the release of certain signals, the end results of which is thickening or rupture of a plaque lining the vessel walls, which in turn leads to the events of coronary artery blockade leading to angina , or carotid artery blockade leading to stroke. So, cholesterol lowering via diet or drugs can therefore be thought of as an anti-inflammatory and plaque stabilizing therapy.
Cholesterol is carried in blood by three major carriers or lipo-proteins : High Density Lipo-proteins (HDL) which means that they contain a lot of the protein portion, which means also that they have high affinity to carry fat, which makes them the good guys who are able to carry fat to the liver away from blood vessels. The second carriers are the Low Density Lipo-proteins (LDL), which means - unlike above - that they are not able to carry much fat away from blood vessels, so their abundance in blood is dangerous and harmful and associated with deposition under the internal linings of blood vessels. The third class of carriers are the Very Low Density Lipo-proteins (VLDL). Most laboratories measure the total cholesterol, total triglycerides (TG) and the HDL fraction, leaving the measurement of the rest of your lipid profile to your doctor, based on the fact that the amount of cholesterol found in the VLDL fraction can be estimated by dividing the triglyceridesby 5, and also based on the fact that LDL fraction can by calculated by subtracting HDL and VLDL from the total cholesterol value.
Total cholesterol is relatively stable over time, and doesn't depend on whether the patient is fasting. However, the TG fraction and HDL vary considerably depending on the fasting status of the patient. That's why the National Cholesterol Education Program (NCEP), Adult Treatment Panel (ATP III) guidelines recommend that only fasting measurements be used to guide management decisions.
Two persons with the same total cholesterol level of 275 mg/dl may have very different lipid profiles. One may have a HDL cholesterol of 110 mg/dl, with a TG of 150 mg/dl, giving an estimated LDL cholesterol of 135 mg/dl. The other may have a HDL cholesterol of 25 mg/dl, with a TG level of 200 mg/dl, giving a LDL cholesterol of 210 mg/dl. Given all other risk factors are equal , the second person would have more than 10-fold higher risk to develop coronary heart disease than the first.
Basic science studies have shown that deposition of cholesterol in arteries is an inflammatory disease. One of the key factors triggering this inflammation is LDL. When LDL is taken up by certain cells in the blood stream, it triggers the release of certain signals, the end results of which is thickening or rupture of a plaque lining the vessel walls, which in turn leads to the events of coronary artery blockade leading to angina , or carotid artery blockade leading to stroke. So, cholesterol lowering via diet or drugs can therefore be thought of as an anti-inflammatory and plaque stabilizing therapy.
Since the advent of statin drugs ( a group of drugs used to lower cholesterol and the most recent of which is crestor ) around 1980s, several studies highlighted their role in reducing the risk of development of cholesterol related diseases on the heart, blood vessels and brain. Five of these studies were specifically significant.
In 1994, the Scandinavian Simvastatin Survival Study (4S) studied 4444 patients for 5.4 years after treating them with 20-40 mg/day of simvastatin. The patients were high risk in terms of both presence of diseases and unfavourable cholesterol profile. The results were a decrease of coronary artery disease deaths by 34%, a decrease in revascularization procedures by 37%, and all-cause death rate by 30%.
In 1995, the West Of Scotland Coronary Prevention Study (WOSCOPS), treated 6569 patients with markedly elevated cholesterol levels with 40 mg/day of pravastatin for 5 years. Coronary events were reduced by 31%. Coronary artery disease deaths were reduced by 28%. Coronary revascularization reduced by 37%, and all-cause deaths reduced by 22%.
In 1996 and 1998, the Cholesterol And Recurrent Events (CARE) study, and the Long term Intervention with Pravastatin in Ischemic Disease (LIPID) study, respectively, showed reduced incidences of bad clinical outcomes too.
In 1994, the Scandinavian Simvastatin Survival Study (4S) studied 4444 patients for 5.4 years after treating them with 20-40 mg/day of simvastatin. The patients were high risk in terms of both presence of diseases and unfavourable cholesterol profile. The results were a decrease of coronary artery disease deaths by 34%, a decrease in revascularization procedures by 37%, and all-cause death rate by 30%.
In 1995, the West Of Scotland Coronary Prevention Study (WOSCOPS), treated 6569 patients with markedly elevated cholesterol levels with 40 mg/day of pravastatin for 5 years. Coronary events were reduced by 31%. Coronary artery disease deaths were reduced by 28%. Coronary revascularization reduced by 37%, and all-cause deaths reduced by 22%.
In 1996 and 1998, the Cholesterol And Recurrent Events (CARE) study, and the Long term Intervention with Pravastatin in Ischemic Disease (LIPID) study, respectively, showed reduced incidences of bad clinical outcomes too.
Another later study was done in 1998. The Air Force / Texas Coronary Atherosclerosis Prevention Study (AFCAPS/TEXCAPS) treated 6605 patients with the usual range of cholesterol for the US population with 20-40 mg/day of lovastatin. Remarkably even in this low risk group of patients, after 5 years of therapy, there was reduced coronary artery disease and death rate by 25%, reduced myocardial infarction events by 40% and decreased revascularization by 33%.
Non-favourable fat profiles are usually detected by routine laboratory screening in an asymptomatic person. That's because except for rare patients whose high cholesterol may present with yellowish skin lesions called xanthomas especially related to eye lids or deposited in tendon bodies like the patellar tendon over the knee joint or the Achilles tendon behind the ankle, the majority of patients have no signs or symptoms of diseae.
200-239 Borderline high.
More than 240 High.
After putting yourself in a category, you've already started yourself on the right track to prevent the unwanted effects of high cholesterol - if present - or to maintain your current healthy cholesterol status, also if present.
On the next post ( 2 of 9 ), we'll continue our advice to you on what to do to save yourself from being pulled into the ever growing maze of not knowing what to do with my lipid profile. Stay tuned.
So, the NCEP guidelines advise that screening for non-favourable lipid profile be done in adults aged 20 years or older with a fasting lipo-protein levels once every 5 years. The profile is categorized based on the LDL, HDL, and total cholesterol values:
LDL Cholesterol (mg/dl)
less than 100 optimal.
100-129 Near optimal.
130-159 Borderline high.
160-189 High.
More than 190 Very high.
HDL Cholesterol (mg/dl)
100-129 Near optimal.
130-159 Borderline high.
160-189 High.
More than 190 Very high.
HDL Cholesterol (mg/dl)
Less than 40 Low.
More than 60 High.
Total cholesterol (mg/dl)
More than 60 High.
Total cholesterol (mg/dl)
Less than 200 desirable.
200-239 Borderline high.
More than 240 High.
After putting yourself in a category, you've already started yourself on the right track to prevent the unwanted effects of high cholesterol - if present - or to maintain your current healthy cholesterol status, also if present.
On the next post ( 2 of 9 ), we'll continue our advice to you on what to do to save yourself from being pulled into the ever growing maze of not knowing what to do with my lipid profile. Stay tuned.
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